Viral evasion of NK-cell activation

被引：19
|
作者：
Rajagopalan, S [1 ]
Long, EO [1 ]
机构：
[1] NIAID, Immunogenet Lab, NIH, Rockville, MD 20852 USA
来源：
TRENDS IN IMMUNOLOGY | 2005年 / 26卷 / 08期
关键词：
D O I：
10.1016/j.it.2005.06.008
中图分类号：
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号：
100102 ;
摘要：
Viruses, such as human cytomegalovirus (HCMV), have developed mechanisms to evade recognition by, and activation of, natural killer (NK) cells, which are cells that provide crucial innate immune defense. Such mechanisms help viruses persist in the human host and attain a state of chronic infection. A recent report documents an antagonistic effect of the main HCMV tegument protein, pp65, on NKp30, a natural cytotoxicity-inducing receptor expressed uniquely on NK cells.
引用
收藏
页码：403 / 405
页数：3
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