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NO vascular control in Duchenne muscular dystrophy
被引:28
|作者:
Crosbie, RH
[1
]
机构:
[1] Univ Calif Los Angeles, Dept Physiol Sci, Los Angeles, CA 90095 USA
关键词:
D O I:
10.1038/83309
中图分类号:
Q5 [生物化学];
Q7 [分子生物学];
学科分类号:
071010 ;
081704 ;
摘要:
A new investigation into Duchenne muscular dystrophy (DMD) pathogenesis suggests that at least part of the muscle degeneration observed in DMD patients may result from the reduced production of muscle membrane-associated neuronal nitric oxide synthase. This reduction may lead to impaired regulation of the vasoconstrictor response and eventual muscle damage.
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页码:27 / 29
页数:3
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