Defining the contribution of aryl hydrocarbon receptor (AhR)-mediated defects in antigen presenting cell (APC) and T cell function that result in impaired clonal expansion and differentiation of influenza virus-specific CD8+ T cells

被引:0
|
作者
Cundiff, JA [1 ]
Neumiller, JJ [1 ]
Lawrence, BP [1 ]
机构
[1] Washington State Univ, Pharmacol Toxicol Program, Pullman, WA 99164 USA
来源
FASEB JOURNAL | 2005年 / 19卷 / 04期
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D O I
暂无
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
引用
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页码:A931 / A931
页数:1
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