Damage-Associated Endogenous TLR4 Ligand Fibronectin-EDA Is Overexpressed in Scleroderma and Drives Persistent Fibrosis Via TLR4 and Inhibition of TLR4 Prevents and Reverses Experimental Dermal Fibrosis: Novel Target for Scleroderma Therapy.

被引:0
|
作者
Bhattacharyya, Swati [1 ]
Tamaki, Zenshiro [1 ]
Wang, Wenxia
Hoover, Paul
Booth, Adam
Dreffs, Alyssa [2 ]
Hinchcliff, Monique E. [1 ]
Fang, Feng
Getsios, Spiro
Yin, Hang [3 ]
White, Eric S. [2 ]
Varga, John [1 ]
机构
[1] Northwestern Univ, Sch Med, 1 2 7, Chicago, IL USA
[2] Univ Michigan, Sch Med, Ann Arbor, MI USA
[3] Univ Colorado, Boulder, CO 80309 USA
来源
ARTHRITIS AND RHEUMATISM | 2012年 / 64卷 / 10期
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
1518
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收藏
页码:S650 / S650
页数:1
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