Spontaneous airway contractions in cultured human fetal lung explants are inhibted by nitric oxide donating drugs.

被引：0

|

作者：

Pandya, H ^{[1
]}

Kotecha, S ^{[1
]}

机构：

[1] Univ Leicester, Dept Child Hlth, Leicester, Leics, England

来源：

AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE | 1999年 / 159卷 / 03期

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中图分类号：

R4 [临床医学];

学科分类号：

1002 ; 100602 ;

摘要：

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页码：A669 / A669

页数：1

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[2] Mutations in T cells from rheumatoid arthritis patients and in a human cell line exposed to nitric oxide donating drugs.
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[3] PATTERNS OF CELLULAR PROLIFERATION AND AIRWAY BRANCHING IN CULTURED FETAL HAMSTER LUNG EXPLANTS
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[4] Endotoxin-induced nitric oxide production rescues airway growth and maturation in atrophic fetal rat lung explants
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[6] Impaired VEGF and nitric oxide signaling after nitrofen exposure in rat fetal lung explants
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[7] Vitamin D inhibits pulmonary surfactant protein gene expression in cultured human fetal lung explants and in pulmonary airway epithelial cells
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[8] Effects of various nitric oxide-donating drugs on adrenergic tension of human seminal vesicles in vitro
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[9] Pre- and postnatal lung development, maturation, and plasticity - Nitric oxide modulates branching morphogenesis in fetal rat lung explants
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[10] Effect of nitric oxide on the development of nitrogen-induced fetal hypoplastic lung explants (pg 17, 2005)
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