p53 isoform delta133p53 in tumor senescence

被引：0
|
作者：
Von Muhlinen, Natalia [1 ]
Beck, Jessica [1 ]
Harris, Curtis C. [1 ]
机构：
[1] NCI, Bethesda, MD 20892 USA
来源：
CANCER RESEARCH | 2018年 / 78卷 / 13期
关键词：
D O I：
10.1158/1538-7445.AM2018-470
中图分类号：
R73 [肿瘤学];
学科分类号：
100214 ;
摘要：
470
引用
收藏
页数：1
相关论文
共 50 条
  • [1] The p53 isoform delta133p53β regulates cancer cell apoptosis in a RhoB-dependent manner
    Arsic, Nikola
    Ho-Pun-Cheung, Alexandre
    Evelyne, Crapez
    Assenat, Eric
    Jarlier, Marta
    Anguille, Christelle
    Colard, Manon
    Pezet, Mikael
    Roux, Pierre
    Gadea, Gilles
    PLOS ONE, 2017, 12 (02):
  • [2] Delta133p53, One of the p53 Isoforms, Suppresses Tumor Cell Proliferation and Induces Cellular Senescence in Hepatocellular Carcinoma Cells
    Nakao, Haruhisa
    Ota, Akinobu
    Ibusuki, Mayu
    Kitano, Rena
    Inoue, Tadahisa
    Kobayashi, Yuuji
    Ohashi, Tomohiko
    Nakade, Yukiomi
    Sumida, Yoshio
    Ito, Kiyoaki
    Yoneda, Masashi
    HEPATOLOGY, 2018, 68 : 1065A - 1065A
  • [3] Targeting Delta133p53 isoform with small-molecule compounds to modulate cellular senescence
    Lissa, Delphine
    Ungerleider, Kyra
    Horikawa, Izumi
    Dranchak, Patricia
    Oliphan, Erin
    Beck, Jessica
    Jo, Sebastien
    Inglese, James
    Harris, Curtis C.
    CLINICAL CANCER RESEARCH, 2020, 26 (12) : 27 - 27
  • [4] Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescence
    Izumi Horikawa
    Kaori Fujita
    Lisa M Miller Jenkins
    Yukiharu Hiyoshi
    Abdul M. Mondal
    Borivoj Vojtesek
    David P. Lane
    Ettore Appella
    Curtis C. Harris
    Nature Communications, 5
  • [5] Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescence
    Horikawa, Izumi
    Fujita, Kaori
    Jenkins, Lisa M. Miller
    Hiyoshi, Yukiharu
    Mondal, Abdul M.
    Vojtesek, Borivoj
    Lane, David P.
    Appella, Ettore
    Harris, Curtis C.
    NATURE COMMUNICATIONS, 2014, 5
  • [6] p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence
    Fujita, Kaori
    Mondal, Abdul M.
    Horikawa, Izumi
    Nguyen, Giang H.
    Kumamoto, Kensuke
    Sohn, Jane J.
    Bowman, Elise D.
    Mathe, A. Mathe
    Schetter, Aaron J.
    Pine, Sharon R.
    Ji, Helen
    Vojtesek, Borivoj
    Bourdon, Jean-Christophe
    Lane, David P.
    Harris, Curtis C.
    CANCER RESEARCH, 2010, 70
  • [7] p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence
    Kaori Fujita
    Abdul M. Mondal
    Izumi Horikawa
    Giang H. Nguyen
    Kensuke Kumamoto
    Jane J. Sohn
    Elise D. Bowman
    Ewy A. Mathe
    Aaron J. Schetter
    Sharon R. Pine
    Helen Ji
    Borivoj Vojtesek
    Jean-Christophe Bourdon
    David P. Lane
    Curtis C. Harris
    Nature Cell Biology, 2009, 11 : 1135 - 1142
  • [8] p53 isoforms Δ133p53 and p53β are endogenous regulators of replicative cellular senescence
    Fujita, Kaori
    Mondal, Abdul M.
    Horikawa, Izumi
    Nguyen, Giang H.
    Kumamoto, Kensuke
    Sohn, Jane J.
    Bowman, Elise D.
    Mathe, Ewy A.
    Schetter, Aaron J.
    Pine, Sharon R.
    Ji, Helen
    Vojtesek, Borivoj
    Bourdon, Jean-Christophe
    Lane, David P.
    Harris, Curtis C.
    NATURE CELL BIOLOGY, 2009, 11 (09) : 1135 - U208
  • [9] Delta133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cells
    Horikawa, Izumi
    Park, Kye-yoon
    Li, Han
    Isogaya, Kazunobu
    Hiyoshi, Yukiharu
    Anami, Katsuhiro
    Robles, Ana I.
    Mondal, Abdul M.
    Fujita, Kaori
    Serrano, Manuel
    Harris, Curtis C.
    CANCER RESEARCH, 2017, 77
  • [10] The p53 isoform delta133p53β regulates cancer cell apoptosis in a RhoB-dependent manner (vol 12, e0172125, 2017)
    Arsic, Nikola
    Ho-Pun-Cheung, Alexandre
    Lopez-Crapez, Evelyne
    Assenat, Eric
    Jarlier, Marta
    Anguille, Christelle
    Colard, Manon
    Pezet, Mikael
    Roux, Pierre
    Gadea, Gilles
    PLOS ONE, 2017, 12 (04):
12345