Profound Hypotension Due to Slow Atrioventricular Nodal Pathway Conduction during Atrial Tachycardia

被引：1

作者：

Nazarian, Saman ^{[1
]}

Dong, Jun ^{[1
]}

Tomaselli, Gordon F. ^{[1
]}

Marine, Joseph E. ^{[1
]}

机构：

[1] Johns Hopkins Univ, Sch Med, Dept Med, Div Cardiol, Baltimore, MD 21205 USA

来源：

PACE-PACING AND CLINICAL ELECTROPHYSIOLOGY | 2009年 / 32卷 / 02期

关键词：

dual atrioventricular nodal physiology; atrial tachycardia; atrioventricular synchrony; hemodynamics; REENTRANT TACHYCARDIA; MANIFESTATIONS; ABLATION; BLOCK;

D O I：

10.1111/j.1540-8159.2008.02216.x

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Paroxysmal supraventricular tachycardia (SVT) may have a variety of hemodynamic effects depending on rate, patient volume status, and presence of structural heart disease or left bundle branch block. We report a case of a patient with atrial tachycardia and dual atrioventricular (AV) nodal physiology who developed profound hypotension during transition from fast to slow AV nodal pathway conduction, despite similar tachycardia cycle length. This case illustrates the potential importance of AV timing in determining the hemodynamic effect of SVT. (PACE 2009; 32:278-280).

引用

页码：278 / 280

页数：3

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