Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug (vol 22, pg 700, 2019)

被引：0
|
作者：
Barak, Boaz
Zhang, Zicong
Liu, Yuanyuan
Nir, Ariel
Trangle, Sari S.
Ennis, Michaela
Levandowski, Kirsten M.
Wang, Dongqing
Quast, Kathleen
Boulting, Gabriella L.
Li, Yi
Bayarsaihan, Dashzeveg
He, Zhigang
Feng, Guoping
机构：
[1] McGovern Institute for Brain Research and Department of Brain & Cognitive Sciences,The School of Psychological Sciences, Faculty of Social Sciences
[2] MIT,The Sagol School of Neuroscience
[3] Tel Aviv University,F.M. Kirby Neurobiology Center, Boston Children’s Hospital and Department of Neurology
[4] Tel Aviv University,Stanley Center for Psychiatric Research
[5] Harvard Medical School,Department of Neurobiology
[6] Broad Institute of MIT and Harvard,Department of Reconstructive Sciences
[7] Harvard Medical School,undefined
[8] University of Connecticut,undefined
来源：
NATURE NEUROSCIENCE | 2019年 / 22卷 / 07期
关键词：
D O I：
10.1038/s41593-019-0441-0
中图分类号：
Q189 [神经科学];
学科分类号：
071006 ;
摘要：
The original and corrected figures are shown in the accompanying Publisher Correction.
引用
收藏
页码：1197 / 1197
页数：1
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  • [1] Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug
    Barak, Boaz
    Zhang, Zicong
    Liu, Yuanyuan
    Nir, Ariel
    Trangle, Sari S.
    Ennis, Michaela
    Levandowski, Kirsten M.
    Wang, Dongqing
    Quast, Kathleen
    Boulting, Gabriella L.
    Li, Yi
    Bayarsaihan, Dashzeveg
    He, Zhigang
    Feng, Guoping
    [J]. NATURE NEUROSCIENCE, 2019, 22 (05) : 700 - +
  • [2] Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug
    Boaz Barak
    Zicong Zhang
    Yuanyuan Liu
    Ariel Nir
    Sari S. Trangle
    Michaela Ennis
    Kirsten M. Levandowski
    Dongqing Wang
    Kathleen Quast
    Gabriella L. Boulting
    Yi Li
    Dashzeveg Bayarsaihan
    Zhigang He
    Guoping Feng
    [J]. Nature Neuroscience, 2019, 22 : 700 - 708
  • [3] Publisher Correction: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug
    Boaz Barak
    Zicong Zhang
    Yuanyuan Liu
    Ariel Nir
    Sari S. Trangle
    Michaela Ennis
    Kirsten M. Levandowski
    Dongqing Wang
    Kathleen Quast
    Gabriella L. Boulting
    Yi Li
    Dashzeveg Bayarsaihan
    Zhigang He
    Guoping Feng
    [J]. Nature Neuroscience, 2019, 22 : 1197 - 1197
  • [4] Neuronal deletion of Gtf2i results in developmental microglial alterations in a mouse model related to Williams syndrome
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    Fischer, Inbar
    Rokach, May
    Elad-Sfadia, Galit
    Shirenova, Sophie
    Ophir, Omer
    Trangle, Sari Schokoroy
    Okun, Eitan
    Barak, Boaz
    [J]. GLIA, 2024, 72 (06) : 1117 - 1135
  • [5] Cognitive-behavioral phenotypes of Williams syndrome are associated with genetic variation in the GTF2I gene, in a healthy population
    Crespi, Bernard J.
    Hurd, Peter L.
    [J]. BMC NEUROSCIENCE, 2014, 15
  • [6] Cognitive-behavioral phenotypes of Williams syndrome are associated with genetic variation in the GTF2I gene, in a healthy population
    Bernard J Crespi
    Peter L Hurd
    [J]. BMC Neuroscience, 15
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