CD36 is not involved in alternative pathway-mediated complement-dependent cytotoxicity on proximal tubular epithelial cells

被引:0
|
作者
Kenawy, Hany Ibrahim [1 ,2 ]
Bevington, Alan [1 ]
Stover, Cordula M. [1 ]
Ali, Youssif M. [1 ,2 ]
Shwaeble, Wilhelm J. [1 ]
Brunskill, Nigel J. [1 ]
机构
[1] Univ Leicester, Dept Infect Immun & Inflammat, Coll Med Biol Sci & Psychol, Leicester, Leics, England
[2] Mansoura Univ, Fac Pharm, Dept Microbiol & Immunol, Mansoura, Egypt
关键词
D O I
10.1016/j.imbio.2012.08.056
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
55
引用
收藏
页码:1148 / 1148
页数:1
相关论文
共 50 条
  • [1] ALTERNATIVE PATHWAY ACTIVATION OF COMPLEMENT BY CULTURED HUMAN PROXIMAL TUBULAR EPITHELIAL-CELLS
    BIANCONE, L
    DAVID, S
    DELLAPIETRA, V
    MONTRUCCHIO, G
    CAMBI, V
    CAMUSSI, G
    [J]. KIDNEY INTERNATIONAL, 1994, 45 (02) : 451 - 460
  • [2] CD36 is involved in high glucose-induced epithelial to mesenchymal transition in renal tubular epithelial cells
    Hou, Yanjuan
    Wu, Ming
    Wei, Jinying
    Ren, Yunzhuo
    Du, Chunyang
    Wu, Haijiang
    Li, Ying
    Shi, Yonghong
    [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2015, 468 (1-2) : 281 - 286
  • [3] Alternative pathway complement activation induces proinflammatory activity in human proximal tubular epithelial cells
    David, S
    Biancone, L
    Caserta, C
    Bussolati, B
    Cambi, V
    Camussi, G
    [J]. NEPHROLOGY DIALYSIS TRANSPLANTATION, 1997, 12 (01) : 51 - 56
  • [4] CD36 is involved in tubular epithelial apoptosis during the progression of diabetic kidney disease.
    Susztak, K
    Ciccone, E
    Wu, D
    Sharma, K
    Bottinger, E
    [J]. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 2003, 14 : 94A - 94A
  • [5] Epithelial-mesenchymal transition confers resistance to complement-dependent cytotoxicity in lung cancer cells
    Goswami, Moloy T.
    Reka, Ajaya K.
    Kurapati, Himabindu
    Kaza, Viritha
    Keshamouni, Venkateshwar G.
    [J]. CANCER RESEARCH, 2012, 72
  • [6] Protein load impairs factor H binding promoting complement-dependent dysfunction of proximal tubular cells
    Buelli, Simona
    Abbate, Mauro
    Morigi, Marina
    Moioli, Daniela
    Zanchi, Cristina
    Noris, Marina
    Zoja, Carla
    Pusey, Charles D.
    Zipfel, Peter F.
    Remuzzi, Giuseppe
    [J]. KIDNEY INTERNATIONAL, 2009, 75 (10) : 1050 - 1059
  • [7] Reduction of complement factor H binding to CLL cells improves the induction of rituximab-mediated complement-dependent cytotoxicity
    S Hörl
    Z Bánki
    G Huber
    A Ejaz
    D Windisch
    B Muellauer
    E Willenbacher
    M Steurer
    H Stoiber
    [J]. Leukemia, 2013, 27 : 2200 - 2208
  • [8] THE SPECIFICITY OF ALTERNATIVE COMPLEMENT PATHWAY-MEDIATED LYSIS OF ERYTHROCYTES - A SURVEY OF COMPLEMENT AND TARGET-CELLS FROM 25 SPECIES
    ISH, C
    ONG, GL
    DESAI, N
    MATTES, MJ
    [J]. SCANDINAVIAN JOURNAL OF IMMUNOLOGY, 1993, 38 (02) : 113 - 122
  • [9] Prenyl Transferases Are Involved in the Regulation of CD20 Levels and Influence Anti-CD20 Monoclonal Antibody-Mediated Activation of Complement-Dependent Cytotoxicity
    Nowis, Dominika
    Winiarska, Magdalena
    Bil, Jacek
    Muchowicz, Angelika
    Wanczyk, Malgorzata
    Glodkowska-Mrowka, Eliza
    Bojarczuk, Kamil
    Firczuk, Malgorzata
    Wilczek, Ewa
    Wachowska, Malgorzata
    Chlebowska, Justyna
    Dwojak, Michal
    Miaczynska, Marta
    Stoklosa, Tomasz
    Golab, Jakub
    [J]. BLOOD, 2011, 118 (21) : 1590 - 1590
  • [10] HIGH GLUCOSE PROMOTES NLRP3 INFLAMMASOME ACTIVATION VIA CD36/ROS PATHWAY IN RENAL TUBULAR EPITHELIAL CELLS
    Hou, Yanjuan
    Wang, Lihua
    [J]. NEPHROLOGY DIALYSIS TRANSPLANTATION, 2019, 34