Evasion from NK cell-mediated immune responses by HIV-1

被引：49
|
作者：
Jost, Stephanie [1 ]
Altfeld, Marcus [1 ]
机构：
[1] Ragon Inst MGH MIT & Harvard, Charlestown, MA 02129 USA
来源：
MICROBES AND INFECTION | 2012年 / 14卷 / 11期
关键词：
HIV-1; NK cells; Escape; Innate immunity; Vaccine; NATURAL-KILLER-CELLS; MHC CLASS-I; IMMUNOGLOBULIN-LIKE RECEPTORS; B-ASSOCIATED TRANSCRIPT-3; VIRUS-INFECTED CELLS; SINGLE-STRANDED RNA; COMPLEX CLASS-I; HLA-B; INHIBITORY RECEPTOR; CUTTING EDGE;
D O I：
10.1016/j.micinf.2012.05.001
中图分类号：
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号：
100102 ;
摘要：
Human immunodeficiency virus type 1 (HIV-1) mostly owes its success to its ability to evade host immune responses. Understanding viral immune escape mechanisms is a prerequisite to improve future HIV-1 vaccine design. This review focuses on the strategies that HIV-1 has evolved to evade recognition by natural killer (NK) cells. (C) 2012 Institut Pasteur. Published by Elsevier Masson SAS. All rights reserved.
引用
收藏
页码：904 / 915
页数：12
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