共 5 条
- [2] Mitochondrial depolarization during TNFalpha-induced apoptosis is caspase-dependent and independent of Bcl-xL and the permeability transition pore. FASEB JOURNAL, 2001, 15 (04): : A169 - A169
- [3] Suppression of endoplasmic reticulum stress-induced caspase activation and cell death by the overexpression of Bcl-xL or Bcl-2 JOURNAL OF BIOCHEMISTRY, 2007, 141 (03): : 401 - 410
- [5] Akt1 targets oxidative stress induced microglial activation and apoptotic vascular injury through modulation of Bcl-xL, mitochondrial membrane potential, and caspase activity FASEB JOURNAL, 2004, 18 (05): : A1034 - A1034