Unveiling the hidden AP-1: revealing the crucial role of AP-1 in ccRCC at single-cell resolution

被引:2
|
作者
Zheng, Jie [1 ,4 ]
Liu, Fengling [3 ,4 ]
Su, Cheng [2 ,4 ]
机构
[1] Guangxi Med Univ, Dept Urol, Affiliated Hosp 1, Nanning, Guangxi, Peoples R China
[2] Guangxi Med Univ, Dept Pediat Surg, Affiliated Hosp 1, Nanning, Guangxi, Peoples R China
[3] Guangxi Med Univ, Dept Gastroenterol, Affiliated Hosp 1, Nanning, Guangxi, Peoples R China
[4] Guangxi Med Univ, Ctr Genom & Personalized Med, Guangxi Collaborat Innovat Ctr Genom & Personalize, Guangxi Key Lab Genom & Personalized Med, Nanning, Peoples R China
关键词
AP-1; Clear cell renal cell carcinoma; scATAC; scRNA; Spatial transcriptomics;
D O I
10.1186/s12943-023-01913-9
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Clear cell renal cell carcinoma (ccRCC), as the most common histological subtype of kidney cancer, has been reported to originate primarily from proximal tubule (PT) cells in the kidney. However, the current research on its associated molecular mechanisms remains relatively limited. In our study, we analyzed multiple single-cell multi-omics datasets obtained from various research teams, revealing the significant role of the activator protein 1 (AP-1) in ccRCC tumorigenesis. The motif activity analysis of transcription factors (TFs) showed a predominant activation of AP-1 in ccRCC cancer cells compared to PT cells. Furthermore, our findings at single-cell resolution revealed a notable absence of AP-1 expression in PT cells when compared to ccRCC cancer cells. In bulk-RNA of discovery cohort, no differential expression of AP-1 was detected in normal kidney and ccRCC samples, which may be attributed to confounding effects in bulk-RNA sequencing. Meanwhile, spatial transcriptomics analysis demonstrated a broader expression range of the AP-1 compared to the ccRCC marker CA9. Moreover, we observed chromatin accessibility of the AP-1 in various cell-types, including PT cells, suggesting that the transcriptional expression of AP-1 in PT cells may be influenced by subsequent transcriptional modifications, reflecting the complex regulatory mechanism of AP-1 transcription. These findings provide important insights for a deeper understanding of the function and regulatory mechanisms of AP-1 in ccRCC, thereby establishing a theoretical foundation for future clinical research and the development of treatment strategies.
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页数:6
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