Friction-Aggravated Skin Disorders-A Review of Mechanism and Related Diseases

被引：0
|
作者：
Hsieh, Chang-Yu [1 ]
Tsai, Tsen-Fang [1 ,2 ]
机构：
[1] Natl Taiwan Univ Hosp, Dept Dermatol, 7 Zhongshan S Rd, Taipei 100, Taiwan
[2] Natl Taiwan Univ, Coll Med, 7 Zhongshan S Rd, Taipei 100, Taiwan
来源：
DERMATITIS | 2023年 / 34卷 / 04期
关键词：
HIDRADENITIS SUPPURATIVA; KOEBNER PHENOMENON; LICHEN-SCLEROSUS; BULLOUS MORPHEA; PATIENT; DERMATITIS; STRETCH; KERATINOCYTES; ACTIVATION; IRRITANT;
D O I：
10.1097/der.0000000000000961
中图分类号：
R75 [皮肤病学与性病学];
学科分类号：
100206 ;
摘要：
Skin is subject to frequent friction injury. Friction affects different structures of the skin, including keratinocytes, melanocytes, fibroblasts, and follicular units. Friction can also stimulate cytokine production. Friction is sensed by the mechanoreceptors, resulting in signal transduction to the nucleus, activating transcription factors and mechanoresponsive genes. Numerous friction-aggravated diseases have been identified, including inflammatory, depositional, follicular, genetic, infectious, and vesiculobullous disorders. Friction, as a potential modifiable aggravator, should be considered when skin diseases are located at friction-prone areas.
引用
收藏
页码：287 / 296
页数：10
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