机构:
Hemostasis Physiology Lab,Xiangya Medical College,Central South UniversityHemostasis Physiology Lab,Xiangya Medical College,Central South University
贺石林
[1
]
范金茹
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机构:
Department of Cardiology,1st Affiliated Hospital,Hunan University of Traditional Chinese Medicine,Changsha 410007,ChinaHemostasis Physiology Lab,Xiangya Medical College,Central South University
范金茹
[2
]
机构:
[1] Hemostasis Physiology Lab,Xiangya Medical College,Central South University
[2] Department of Cardiology,1st Affiliated Hospital,Hunan University of Traditional Chinese Medicine,Changsha 410007,China
Cerebrocardiac thrombotic disease such as acute my-ocardial infarction and acute ischemic stroke cause significant morbidity and mortality. Present therapies for these diseases besides anttcoagulation and fibrinolysis mainly aim toward limiting platelet adhesion and aggregation processes. However, their incomplete effectiveness suggests that other mechanisms may be important. Although the underlying cellular and molecular mechanisms of