共 50 条
T-cell receptor architecture and clonal tiding provide insight into the transformation trajectory of peripheral T-cell lymphomas
被引:2
|作者:
Willscher, Edith
[1
]
Schultheiss, Christoph
[2
,3
]
Paschold, Lisa
[1
]
Schuemann, Franziska Lea
[1
]
Schmidt-Barbo, Paul
[2
]
Thiele, Benjamin
[2
,3
]
Bauer, Marcus
[4
]
Wickenhauser, Claudia
[4
]
Weber, Thomas
Binder, Mascha
[2
,3
]
机构:
[1] Martin Luther Univ Halle Wittenberg, Internal Med 4, Oncol Hematol, Halle, Germany
[2] Univ Basel, Dept Biomed Translat Immuno Oncol, Basel, Switzerland
[3] Univ Hosp Basel, Div Med Oncol, Basel, Switzerland
[4] Martin Luther Univ Halle Wittenberg, Dept Pathol, Halle, Germany
关键词:
TRANSCRIPTION FACTORS;
REPERTOIRE;
TET2;
DIFFERENTIATION;
MANAGEMENT;
DISEASE;
DNMT3A;
D O I:
10.3324/haematol.2024.285395
中图分类号:
R5 [内科学];
学科分类号:
1002 ;
100201 ;
摘要:
While T-cell lymphomas are classified as mature neoplasms, emerging evidence indicates that malignant transformation may occur at an earlier stage of T-cell maturation. In this study, we determined clonal architecture in a broad range of T-cell lymphomas. Our multidimensional profiling indicates that many of these lymphomas do in fact emerge from an immature lymphoid T-cell precursor at a maturation stage prior to V(D)J rearrangement that undergoes branching evolution. Consequently, at single-cell resolution we observed considerable clonal tiding under selective therapeutic pressure. T-cell receptor next-generation sequencing suggested a highly biased usage of TRBV20-1 gene segments as part of multiple antigen receptor rearrangements per patient. The predominance of TRBV20-1 was found across all major T-cell lymphoma subtypes analyzed. This suggested that this particular V gene - independently of complementarity-determining region 3 configuration - may represent a driver of malignant transformation. Together, our data indicate that T-cell lymphomas are derived from immature lymphoid precursors and display considerable intratumoral heterogeneity that may provide the basis for relapse and resistance in these hard-to-treat cancers.
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页码:457 / 469
页数:13
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