Ethidium bromide-induced inhibition of mitochondrial gene transcription suppresses glucose-stimulated insulin release in the mouse pancreatic β-cell line βHC9

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J Biol Chem | / 32卷 / 20300期
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  • [1] Ethidium bromide-induced inhibition of mitochondrial gene transcription suppresses glucose-stimulated insulin release in the mouse pancreatic β-cell line βHC9
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