An evolutionarily conserved AnkyrinG-dependent motif clusters axonal K2P K+ channels

被引:1
|
作者
Escobedo, Gabriel [1 ]
Wu, Yu [1 ]
Ogawa, Yuki [1 ]
Ding, Xiaoyun [1 ]
Rasband, Matthew N. [1 ]
机构
[1] Baylor Coll Med, Dept Neurosci, Houston, TX 77030 USA
来源
JOURNAL OF CELL BIOLOGY | 2024年 / 223卷 / 10期
基金
美国国家卫生研究院;
关键词
POTASSIUM CHANNELS; INITIAL SEGMENT; SODIUM-CHANNELS; SPECTRIN; MEMBRANE; BINDING; FAMILY; TREK-1; TRAAK;
D O I
10.1083/jcb.202401140
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
The evolution of ion channel clustering at nodes of Ranvier enabled the development of complex vertebrate nervous systems. At mammalian nodes, the K+ leak channels TRAAK and TREK-1 underlie membrane repolarization. Despite the molecular similarities between nodes and the axon initial segment (AIS), TRAAK and TREK-1 are reportedly node-specific, suggesting a unique clustering mechanism. However, we show that TRAAK and TREK-1 are enriched at both nodes and AIS through a common mechanism. We identified a motif near the C-terminus of TRAAK that is necessary and sufficient for its clustering. The motif first evolved among cartilaginous fish. Using AnkyrinG (AnkG) conditional knockout mice, CRISPR/Cas9-mediated disruption of AnkG, co-immunoprecipitation, and surface recruitment assays, we show that TRAAK forms a complex with AnkG and that AnkG is necessary for TRAAK's AIS and nodal clustering. In contrast, TREK-1's clustering requires TRAAK. Our results expand the repertoire of AIS and nodal ion channel clustering mechanisms and emphasize AnkG's central role in assembling excitable domains.
引用
收藏
页数:13
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