EFFECTS OF THIO-GROUP MODIFICATION AND CA2+ ON AGONIST-SPECIFIC STATE TRANSITIONS OF A CENTRAL NICOTINIC ACETYLCHOLINE-RECEPTOR

被引：36
|
作者：
LUKAS, RJ
MORIMOTO, H
BENNETT, EL
机构：
来源：
BIOCHEMISTRY | 1979年 / 18卷 / 11期
关键词：
D O I：
10.1021/bi00578a038
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
引用
收藏
页码：2384 / 2395
页数：12
相关论文
共 11 条
  • [1] EFFECTS OF THIOL MODIFICATION AND CA++ ON AGONIST-SPECIFIC STATE TRANSITIONS OF NICOTINIC ACETYLCHOLINE-RECEPTOR FROM TORPEDO-CALIFORNICA ELECTROPLAX
    MILLER, JV
    LUKAS, RJ
    BENNETT, EL
    LIFE SCIENCES, 1979, 24 (20) : 1893 - 1899
  • [2] EFFECTS OF THIO-GROUP MODIFICATIONS OF TORPEDO-CALIFORNICA ACETYLCHOLINE-RECEPTOR ON ION FLUX ACTIVATION AND INACTIVATION KINETICS
    WALKER, JW
    RICHARDSON, CA
    MCNAMEE, MG
    BIOCHEMISTRY, 1984, 23 (11) : 2329 - 2338
  • [3] EFFECTS OF THIO-GROUP MODIFICATIONS ON THE ION PERMEABILITY CONTROL AND LIGAND-BINDING PROPERTIES OF TORPEDO-CALIFORNICA ACETYLCHOLINE-RECEPTOR
    WALKER, JW
    LUKAS, RJ
    MCNAMEE, MG
    BIOCHEMISTRY, 1981, 20 (08) : 2191 - 2199
  • [4] DIABETIC STATE-INDUCED RAPID INACTIVATION OF NONCONTRACTILE CA2+ MOBILIZATION OPERATED BY NICOTINIC ACETYLCHOLINE-RECEPTOR IN MOUSE DIAPHRAGM MUSCLE
    KIMURA, I
    TSUNEKI, H
    DEZAKI, K
    KIMURA, M
    BRITISH JOURNAL OF PHARMACOLOGY, 1995, 116 (06) : 2685 - 2690
  • [5] Agonist-Specific Pharmacological Effects of CMPI and NS9283 at (α4) 3(β2)2 Neuronal Nicotinic. Acetylcholine Receptors
    Munoz, Kemburli
    Deba, Farah
    Hamouda, Ayman K.
    BIOPHYSICAL JOURNAL, 2018, 114 (03) : 296A - 296A
  • [6] EFFECTS OF LIPIDS ON ACETYLCHOLINE-RECEPTOR - ESSENTIAL NEED OF CHOLESTEROL FOR MAINTENANCE OF AGONIST-INDUCED STATE TRANSITIONS IN LIPID VESICLES
    CRIADO, M
    EIBL, H
    BARRANTES, FJ
    BIOCHEMISTRY, 1982, 21 (15) : 3622 - 3629
  • [7] Distinct effects of omega-toxins and various groups of Ca2+-entry inhibitors on nicotinic acetylcholine receptor and Ca2+ channels of chromaffin cells
    Villarroya, M
    DelaFuente, MT
    Lopez, MG
    Gandia, L
    Garcia, AG
    EUROPEAN JOURNAL OF PHARMACOLOGY, 1997, 320 (2-3) : 249 - 257
  • [8] Central nicotinic acetylcholine receptor involved in Ca2+-calmodulin-endothelial nitric oxide synthase pathway modulated hypotensive effects
    Cheng, Pei-Wen
    Lu, Pei-Jung
    Chen, Siang-Ru
    Ho, Wen-Yu
    Cheng, Wen-Han
    Hong, Ling-Zong
    Yeh, Tung-Chen
    Sun, Gwo-Ching
    Wang, Ling-Lin
    Hsiao, Michael
    Tseng, Ching-Jiunn
    BRITISH JOURNAL OF PHARMACOLOGY, 2011, 163 (06) : 1203 - 1213
  • [9] T cells express α7-nicotinic acetylcholine receptor Subunits that require a functional TCR and leukocyte-specific protein tyrosine kinase for nicotine-induced Ca2+ response
    Razani-Boroujerdi, Seddigheh
    Boyd, R. Thomas
    Davila-Garcia, Martha I.
    Nandi, Jayashree S.
    Mishra, Neerad C.
    Singh, Shashi P.
    Pena-Philippides, Juan Carlos
    Langley, Raymond
    Sopori, Mohan L.
    JOURNAL OF IMMUNOLOGY, 2007, 179 (05): : 2889 - 2898
  • [10] Pregnancy-enhanced store-operated Ca2+ channel function in uterine artery endothelial cells is associated with enhanced agonist-specific transient receptor potential channel 3-inositol 1,4,5-trisphosphate receptor 2 interaction
    Gifford, Shannon M.
    Yi, Fu-Xian
    Bird, Ian M.
    JOURNAL OF ENDOCRINOLOGY, 2006, 190 (02) : 385 - 395
12